This is a common problem with pellets. If you go looking for forums on the matter, you will see that it is rather typical for patients to have a similar response somewhere around the 4th and 5th implantation. This stems from poor oversight/management, and design flaws in the pellets themselves. Keep in mind that all pellets are made the same way. This may seem beneficial at first glance, however, what this really means is that “6 months” worth of medication dissolves in a way that sends your levels far too high in the first month, and then plummeting far too fast and low in the next 2-3.
For the athlete looking for an edge a dosing of 20mg per day will generally prove to be the minimal with 40mg per day being far more optimal. While 40mg per day will provide a nice boost in-terms of overall athletic performance if you’re really looking to transform your physique you will probably need a far greater dose. As this steroid will not provide massive amounts of lean tissue most bodybuilders will not mess with it and if they do they will necessarily take massive amounts making it a poor choice for off-season periods of growth. The dieting bodybuilder however might find a more suitable use for the steroid but again there are more efficient choices for this individual. In either case, as it is an anabolic steroid that is hepatic total use should not extend past the 8 week mark but many will find 6 weeks to be just about perfect.
For the female Turinabol user 5mg per day would be the starting point with 10mg per day being the absolute max . Most females will need to start at 5mg per day to see how they react but understand if you approach the 10mg mark you will increase the probability of virilization. While 5mg per day may not sound like much it is important to remember on a per milligram basis Turinabol appears to be much stronger in women than it is in men meaning lower doses will have a far reaching and pronounced affect.
A 31-year-old man presenting with an 18-month history of sexual dysfunction resulting from severe adult-onset IHH (LH U/L, FSH U/L, T nmol/L). Initial therapy with 50 mg of clomiphene citrate (CC) three times a day for 7 days, with overnight LH pulse profiling and 9 am T levels evaluated at baseline and on completion. A 2-month washout period, followed by low-dose maintenance therapy (25-50 mg/d) for 4 months.
MAIN OUTCOME MEASURE(S):Baseline and stimulated T levels and LH pulsatility; effect on sexual function.
RESULT(S):Clomiphene therapy resulted in complete normalization of pulsatile gonadotropin secretion, serum T level, and sexual function. CONCLUSION(S):Isolated hypogonadotropic hypogonadism may result from an acquired defect of enhanced hypothalamic sensitivity to E-mediated negative feedback. Whereas direct T replacement therapy can further suppress endogenous gonadotropin secretion, treating IHH men with gonadotropins can stimulate endogenous T secretion and enhance fertility potential. On theoretical grounds, reversal of gonadotropin deficiency with CC might be expected to have a similar biological effect.